The high failure rate of rotator cuff repair is often attributed to the fact that a normal zonal insertion site is not regenerated between tendon and bone. [1,2] Interestingly, a previous study in our laboratory demonstrated that a patellar tendon autograft (PTA) used to repair a central-third patellar tendon (PT) defect also does not regenerate a normal zonal insertion site at 12 weeks following surgery. Therefore, using our model of tendon healing, our objective was to design a biologic augmentation (BA) that could be implemented at the insertion site between tendon and bone to help promote integration. The results of this study have potential application in developing new techniques for rotator cuff repair.

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