Abdominal aortic aneurysm (AAA) rupture occurs when wall stress exceeds wall strength. Endografts aim to prevent rupture by reducing wall stress. However, after implantation, AAA may shrink, grow, or stabilize in diameter, even in the absence of apparent flow into the sac or pressurization. This may result in graft failure through loss of seal, kinking, or component separation. Insight from models of treated AAA could provide insight into treatment methods/limitations and for device design. The nature of the stress reduction in AAA by endograft treatment was modeled using FEM in two patients followed at three time points each (pre-treatment, early follow-up, and late follow-up) — one with a “stable” aneurysm and one with a “shrinking” aneurysm.
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ASME 2009 Summer Bioengineering Conference
June 17–21, 2009
Lake Tahoe, California, USA
Conference Sponsors:
- Bioengineering Division
ISBN:
978-0-7918-4891-3
PROCEEDINGS PAPER
FEM Modeling of Patient-Specific AAA Treated With Endografts
Clark A. Meyer,
Clark A. Meyer
Texas A&M University, College Station, TX
Search for other works by this author on:
James E. Moore, Jr.
James E. Moore, Jr.
Texas A&M University, College Station, TX
Search for other works by this author on:
Clark A. Meyer
Texas A&M University, College Station, TX
James E. Moore, Jr.
Texas A&M University, College Station, TX
Paper No:
SBC2009-206444, pp. 527-528; 2 pages
Published Online:
July 19, 2013
Citation
Meyer, CA, & Moore, JE, Jr. "FEM Modeling of Patient-Specific AAA Treated With Endografts." Proceedings of the ASME 2009 Summer Bioengineering Conference. ASME 2009 Summer Bioengineering Conference, Parts A and B. Lake Tahoe, California, USA. June 17–21, 2009. pp. 527-528. ASME. https://doi.org/10.1115/SBC2009-206444
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