The link between hemodynamic forces, notably wall shear stress (WSS), and atherogenesis is well established. Patient-specific computational fluid dynamics (CFD) modeling of human vasculature has attracted recent attention because it allows investigators to determine areas at risk for plaque formation and subsequent rupture [1]. Non-invasive in vivo imaging methods such as magnetic resonance (MR) angiography allow acquisition of vascular geometry and cross-sectional velocity such that a CFD model can determine the spatial distribution of WSS. WSS may then be correlated with phenomena such as wall thickening.

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